WDR4 Rabbit Polyclonal Antibody (CAB16487)
- SKU:
- CAB16487
- Product Type:
- Antibody
- Reactivity:
- Human
- Rat
- Host Species:
- Rabbit
- Isotype:
- IgG
- Antibody Type:
- Polyclonal Antibody
- Research Area:
- Epigenetics and Nuclear Signaling
Frequently bought together:
Description
Product Name: | WDR4 Rabbit Polyclonal Antibody |
SKU: | CAB16487 |
Size: | 20uL, 100uL |
Isotype: | IgG |
Host Species: | Rabbit |
Reactivity: | Human,Rat |
Immunogen: | Recombinant fusion protein containing a sequence corresponding to amino acids 20-250 of human WDR4 (NP_061139.2). |
Sequence: | SRFL ATSI ASSD DDSL FIYD CSAA EKKS QENK GEDA PLDQ GSGA ILAS TFSK SGSY FALT DDSK RLIL FRTK PWQC LSVR TVAR RCTA LTFI ASEE KVLV ADKS GDVY SFSV LEPH GCGR LELG HLSM LLDV AVSP DDRF ILTA DRDE KIRV SWAA APHS IESF CLGH TEFV SRIS VVPT QPGL LLSS SGDG TLRL WEYR SGRQ LHCC HLAS LQEL VDPQ APQK FAAS RIA |
Tested Applications: | WB ELISA |
Recommended Dilution: | WB,1:500 - 1:2000 |
Synonyms: | hWH; Wuho; MIGSB; TRM82; GAMOS6; TRMT82; WDR4 |
Positive Sample: | HeLa,HepG2,Rat lung |
Conjugate: | Unconjugated |
Cellular Localization: | Nucleus. |
Calculated MW: | 45kDa |
Observed MW: | 45kDa |
This gene encodes a member of the WD repeat protein family. WD repeats are minimally conserved regions of approximately 40 amino acids typically bracketed by gly-his and trp-asp (GH-WD), which may facilitate formation of heterotrimeric or multiprotein complexes. Members of this family are involved in a variety of cellular processes, including cell cycle progression, signal transduction, apoptosis, and gene regulation. This gene is excluded as a candidate for a form of nonsyndromic deafness (DFNB10), but is still a candidate for other disorders mapped to 21q22.3 as well as for the development of Down syndrome phenotypes. Alternatively spliced transcript variants have been found for this gene.
Purification Method: | Affinity purification |
Gene ID: | 10785 |
Storage Buffer: | Store at -20℃. Avoid freeze / thaw cycles.Buffer: PBS with 0.01% thimerosal,50% glycerol,pH7.3. |