The SLC39A9 Antibody (PAC022851) is a valuable tool for researchers studying SLC39A9, a critical protein involved in cellular zinc transport. This polyclonal antibody, produced in rabbits, exhibits high reactivity with human samples and is suitable for Western blot applications. By specifically binding to the SLC39A9 protein, this antibody enables precise detection and analysis in a variety of cell types, making it an essential component in studies related to cellular zinc homeostasis and its impact on various biological processes.SLC39A9, also known as ZIP9, plays a crucial role in maintaining optimal zinc levels within cells, which is essential for numerous cellular functions including growth, development, and immune response.
Dysregulation of zinc transport mediated by SLC39A9 has been implicated in diseases such as cancer, neurodegenerative disorders, and diabetes. Therefore, understanding the role of SLC39A9 in cellular zinc homeostasis is key to unraveling its contribution to disease pathogenesis and exploring potential therapeutic interventions.By utilizing the SLC39A9 Antibody (PAC022851), researchers can gain valuable insights into the function of SLC39A9 in health and disease, paving the way for the development of targeted therapies aimed at modulating cellular zinc levels for therapeutic benefit.
Antibody Name:
SLC39A9 Antibody (PACO22851)
Antibody SKU:
PACO22851
Size:
100ul
Host Species:
Rabbit
Tested Applications:
ELISA, IHC
Recommended Dilutions:
ELISA:1:2000-1:10000, IHC:1:50-1:100
Species Reactivity:
Human, Mouse, Rat
Immunogen:
Synthetic peptide corresponding to a region derived from internal residues of human solute carrier family 39, member 9
Form:
Liquid
Storage Buffer:
Rabbit IgG in pH7.3 PBS, 0.05% NaN3, 50% Glycerol.
Purification Method:
Antigen Affinity Purified
Clonality:
Polyclonal
Isotype:
IgG
Conjugate:
Non-conjugated
Immunohistochemical analysis of paraffin-embedded Human breast cancer tissue using at dilution 1/20.
Background:
ZIP9 is believed to function as a zinc homeostasis regulator acting in the secretory pathway. This process seems to not significantly alter cytosolic zinc homeostasis.
