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Recombinant HER2 Monoclonal Antibody (CPAB0212)

SKU:
CPAB0212
Product Type:
Antibody
Antibody Type:
Monoclonal Antibody
Reactivity:
Human
€149
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Description

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Recombinant HER2 Monoclonal Antibody (CPAB0212)

The Recombinant HER2 Antibody (CPAB0212) is a cutting-edge tool for researchers studying the HER2 protein, a critical biomarker in cancer biology. This highly specific antibody, produced through recombinant technology, offers unparalleled sensitivity and accuracy in detecting HER2 in various sample types. HER2, also known as human epidermal growth factor receptor 2, is a key player in cell growth and division, and its dysregulation is commonly associated with aggressive forms of cancer, particularly breast cancer. The Recombinant HER2 Antibody enables precise visualization and quantification of HER2 expression levels, providing valuable insights into disease progression and treatment response.

Whether investigating HER2 signaling pathways, evaluating therapeutic efficacy, or exploring novel treatment strategies, the Recombinant HER2 Antibody is an essential tool for researchers in the field of oncology. Its superior performance in immunohistochemistry, flow cytometry, and other analytical techniques make it a versatile option for a wide range of experimental settings. Dive deeper into HER2 biology with confidence and precision using the Recombinant HER2 Antibody (CPAB0212).

Product Name:Recombinant HER2 Antibody
Product Sku:CPAB0212
Size:1mg
Host Species:
Immunogen:
Clone:
Reactivity:Human
Applications:
Purification Method:
Isotype:
Background:HER-2/neu (erbB-2) encodes an 185-kDa orphan receptor tyrosine kinase that is constitutively active as a dimer and displays potent oncogenic activity when overexpressed. Herstatin, as the product of alternative HER-2 transcript, retains intron 8. The herstatin mRNA is expressed in normal human fetal kidney and liver, but is at reduced levels relative to p185HER-2 mRNA in carcinoma cells that contain an amplified HER-2 gene. Herstatin appears to be an inhibitor of p185HER-2, because it disrupts dimers, reduces tyrosine phosphorylation of p185, and inhibits the anchorage-independent growth of transformed cells that overexpress HER-2.
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