Phospho-Erk1-T202/Y204 + Erk2-T185/Y187 Rabbit Monoclonal Antibody (CABP0974)
- SKU:
- CABP0974
- Product Type:
- Antibody
- Reactivity:
- Human
- Mouse
- Rat
- Host Species:
- Rabbit
- Isotype:
- IgG
- Antibody Type:
- Monoclonal Antibody
Frequently bought together:
Description
Product Name: | Phospho-Erk1-T202/Y204 + Erk2-T185/Y187 Rabbit Monoclonal Antibody |
SKU: | CABP0974 |
Size: | 20uL, 100uL |
Isotype: | IgG |
Host Species: | Rabbit |
Reactivity: | Human,Mouse,Rat |
Immunogen: | A synthetic phosphorylated peptide around T202 & Y204 of human ERK1 (P27361). |
Sequence: | LTEY V |
Tested Applications: | WB IHC-P IF/ICC ELISA |
Recommended Dilution: | WB,1:500 - 1:2000 IHC-P,1:50 - 1:200 IF/ICC,1:50 - 1:200 |
Synonyms: | ERK-1; ERK1; ERT2; HS44KDAP; HUMKER1A; P44ERK1; P44MAPK; PRKM3; p44-ERK1; p44-MAPK; Phospho-ERK1-T202/Y204 + ERK2-T185/Y187 |
Positive Sample: | 293T+ATP,NIH/3T3+ATP,C6+PMA/TPA |
Conjugate: | Unconjugated |
Cellular Localization: | caveola, cytoplasm, cytoskeleton, cytosol, early endosome, endoplasmic reticulum lumen, focal adhesion, Golgi apparatus, late endosome, mitochondrion, nuclear envelope, nucleoplasm, nucleus, plasma membrane |
Calculated MW: | 42kDa/44kDa |
Observed MW: | 42kDa/44kDa |
The protein encoded by this gene is a member of the MAP kinase family. MAP kinases, also known as extracellular signal-regulated kinases (ERKs), act in a signaling cascade that regulates various cellular processes such as proliferation, differentiation, and cell cycle progression in response to a variety of extracellular signals. This kinase is activated by upstream kinases, resulting in its translocation to the nucleus where it phosphorylates nuclear targets. Alternatively spliced transcript variants encoding different protein isoforms have been described. [provided by RefSeq, Jul 2008]
Purification Method: | Affinity purification |
Gene ID: | 5595/5594 |
Clone Number: | ARC1595 |
Storage Buffer: | Store at -20℃. Avoid freeze / thaw cycles.Buffer: PBS with 0.05% proclin300,0.05% BSA,50% glycerol,pH7.3. |