The FAM19A3 Polyclonal Antibody (PAC052506) is a valuable tool for researchers studying FAM19A3, a secreted protein with potential roles in various biological processes. Raised in rabbits, this antibody is highly specific for human samples and has been validated for use in Western blot applications. By binding to the FAM19A3 protein, this antibody enables the detection and analysis of FAM19A3 in a range of cell types, making it ideal for studies in molecular biology and disease research.FAM19A3, also known as TAFA3, is a member of the TAFA family of proteins and has been implicated in neuronal development and function, as well as potential roles in immune response regulation.
Its molecular functions and potential involvement in disease pathology make it a promising target for research into neurological disorders, neuroinflammation, and other related conditions. Understanding the role of FAM19A3 could provide valuable insights into disease mechanisms and identify novel therapeutic strategies for these conditions.
Antibody Name:
FAM19A3 Antibody (PACO52506)
Antibody SKU:
PACO52506
Size:
50ug
Host Species:
Rabbit
Tested Applications:
ELISA
Recommended Dilutions:
Species Reactivity:
Human
Immunogen:
Recombinant Human Protein FAM19A3 protein (31-169AA)
Protein FAM19A3 (Chemokine-like protein TAFA-3), FAM19A3, TAFA3
UniProt Protein Function:
Plays a role in the regulation of microglia polarization.
UniProt Protein Details:
NCBI Summary:
This gene is a member of the TAFA family which is composed of five highly homologous genes that encode small secreted proteins. These proteins contain conserved cysteine residues at fixed positions, and are distantly related to MIP-1alpha, a member of the CC-chemokine family. The TAFA proteins are predominantly expressed in specific regions of the brain, and are postulated to function as brain-specific chemokines or neurokines, that act as regulators of immune and nervous cells. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene. [provided by RefSeq, Nov 2011]